194. Is there evidence of enhanced duodenogastric reflux in Barrett's esophagus?
R.C. Heading (Edinburgh)

195. What is the effect of gastric juice on Barrett's mucosa?
RM. Bremner, T.R. DeMeester (Los Angeles)

196. Is the mucosa in the CLE usually the site of acid hypersecretion?
C.G. Bremner, S.F. Hoeft (Los Angeles)

197. What is the pH measurement profile in patients with CLE?
S. Bruley des Varannes, J.P. Galmiche (Nantes), C. Scarpignato (Parma)

198. What is the mean distal sphincteric pressure in patients with CLE?
C. Iascone, A. Moraldi, D. Castiglia (Rome)

199. Does esophageal dysfunction in CLE influence bolus propulsion through the esophagus? Does it differ from that of reflux esophagitis?
HJ. Stein (Munich), T.R. DeMeester (Los Angeles)

200. Do the etiologic factors in disorders of esophageal motility suggest that regression is no longer possible?
T.R. DeMeester (Los Angeles)

201. What are the possible reasons for the differing results of tests of gastric emptying?
G.G. Jamieson (Adelaide)

202. Should the use of pharmacologic agents promoting gastric emptying be reconsidered?
R. W. McCallum (Charlottesville)

203. Can the development of Barrett's mucosa be found after esophagogastrostomy?
P.M. Cugnenc, Ph. Wind, M. Bouchoucha, J.Ph. Barbier