Primary Motility  Disorders of the  Esophagus
 The Esophageal
 Esophagogastric  Junction

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Volume: The Esophageal Mucosa
Chapter: Mucosa and histology

208. Are parietal and chief cells present in Barrett's esophagus, and are such cells functional?
D.A. Antonioli (Boston)

209. Does such a finding explain that Barrett's ulcers do not respond to conservative medical regimen?
G. Delpre (Petah Tikva),

210. Are there histologic differences between Barrett's mucosa of long segment (over 3 cm above the LES) and short segment (3 cm or less above the LES)?
S.R. Hamilton (Baltimore)

211. Is the presence of specialized epithelium necessary for the diagnosis of Barrett's esophagus?
H.D. Appelman (Ann Arbor)

212. Apart from the specialized mucosal type, how can Barrett's mucosa be distinguished from the normal cardia mucosa?
H.D. Appelman (Ann Arbor)

213. Are all different types of Barrett's epithelium originally present, or do they appear only after some period of evolution?
D. Parekh, G.W.B. Clark, T.R. DeMeester (Los Angeles)

214. Does Barrett's mucosa have the same histologic features in adults and children?
D.A. Antonioli (Boston)

215. Which epithelial differentiations predispose to dysplasia and carcinoma?
F. Potet (Paris)

216. Does columnar metaplasia ever derive from a proliferation of the submucosal glands or their ducts, after chronic reflux has destroyed the squamous mucosa?
P. Keeling (Dublin)

217. Can Barrett's mucosa develop following an injury other than reflux, such as irradiation or chemotherapy?
W.V. Bogomoletz (Reims)

218. Are Barrett's mucosa and the neoplastic colonic mucosa evidence of a diffuse gastrointestinal inflammatory reaction?
A. Keshavarzian, SJ. Sontag (Hines)

219. Does Helicobacter pylori colonize the gastric metaplasia of CLE and induce additional disease in that site?
P. Vincent (Lille)

Publication date: May 1994 OESO©2015