7th Congress home page
Medical Treatments (Cont.)
Distance Learning in Healthcare and Gastroenterology
Interactive, multi-disciplinary discussion on a clinical case
September 3, 2003
Presidents: S.J. SPECHLER
What are the compared deleterious effects of bile acids and trypsin concentration in the refluxate of animal models?
Does reflux of duodenal content induce glandular metaplasia in animal models?
Can the possibility of Barrett metaplasia developing from the stem cells in the esophageal basal layer be envisaged consistent with experimental duodeno-esophageal reflux?
Barrett’s esophagus: is there a clinically useful animal model at all?
(Rochester - MN)
What is the prevalence of Barrett’s esophagus after total gastrectomy?
What is the prevalence of alkaline material in Barrett patients?
Compared to patients with GERD, what is the spectrum of exposure of esophagus to bilirubin in Barrett patients?
Can the respective roles of acid and bile in the development of Barrett’s esophagus be assessed? Should a synergistic action be envisaged?
Is the DGR Bilitec profile different in GERD patients with, and without Barrett’s esophagus?
Is there a different bilirubin exposure in long segment compared to short segment Barrett’s esophagus?
Is there a greater bilirubin exposure time in patients with complicated Barrett’s esophagus?
What is the relationship between carditis and Barrett metaplasia?
R. GENTA, E.R. TSCHANZ
What is the respective influence of reflux and H pylori in the development of carditis?
Can a prophylactic effect of medical therapy on the development of Barrett’s esophagus be envisaged?
Can a specific effect of PPIs on the development of dysplasia be explained?
Is there a place for a non endoscopic surveillance of these patients?
R.E. SAMPLINER (Usa)
PRESENTATION OF SELECTED POSTERS