7th Congress home page
Scientific Committee
Scientific Program


PS1 Physiology
PS2 Pathophysiology
PS3 Investigations
PS4 Investigations (Cont.)

PS5 Extra-esophageal symptoms
...........ENT symptoms

PS6 Pulmonary symptoms
PS7 Histology
...........Medical Treatments

PS4 Investigations (Cont.) Monday, September 1, 2003
Presidents: D.O. CASTELL (USA)


3:30 p.m.
How should the variability in the composition of DGR, and especially the lack of assessing the pancreatic component, be interpreted?
A. SMYTHE (Sheffield)

What is the in vivo reliability of the Bilitec sensor for the detection of duodenal reflux through the foregut?

Which is the most appropriate Bilitec wave length for detection of bilirubin in the stomach?
J. TACK (Leuven)

Does concentration of bilirubin, as evaluated by Bilitec, correlate with that of bile acids and trypsin?
A. PAPAZIAN (Amiens)

Is bilirubin, as assessed by Bilitec, a good tracer for duodenal reflux into the esophagus?
A. SMYTHE (Sheffield)

What is the level of sensitivity and reliability of Bilitec at low pH values and high bilirubin concentrations?
P. BECHI (Florence)

4:00 p.m.
4:10 p.m.

I. HERRMANN (Italy) - N. DI MARTINO (Italy)
4:10 p.m.
Topic Forum
What is the value of the multi-channel intra-luminal impedance monitoring in the assessment of non-acid GER?
G.W. FALK (Cleveland) M. VAEZI (Cleveland)
4:20 p.m.

4:25 p.m.
4:40 p.m.

Key note address:
Multichannel intraluminal impedance. The futur of esophageal function testing.
D.O. CASTELL(Charleston)

What is the percentage of GERD patients with exclusive esophageal symptoms?
G.R. LOCKE (Rochester-MN)

What is the prevalence of DGER in patients with GERD?
M. FEIN (Wurzburg)

What are the symptoms related to non-acid reflux? At what stage do they occur?
S. HARDING (Birmingham-AL)

Can a primary pathologic DGR be identified with Bilitec in GERD patients with gastric symptoms?

Does a positive test indicate a relationship between symptoms and acidic reflux?
G. NARDONE (Naples)


5:05 p.m.
Topic Forum
Extra-digestive manifestations of DGER and control of nocturnal acid secretion.
R.W. Mc CALLUM (Kansas City) - F.MION (Lyons)
5:15 p.m.


  What are the circadian variations of intra-gastric acid, pepsin and bile acids content in patients with reflux esophagitis?
D.NEHRA (Carshalton)

Why do patients with H pylori, in spite of a higher intra-gastric pH, have a more severe esophageal acid exposure?
M. BLASER (New York)

Is antro-duodenal motility altered after cholecystectomy?
T.N. WALSH (Dublin)

5:35 p.m.
5:40 p.m.

5:40 p.m